When it comes to serum Creatinine, most nephropathy patients think of uremia first. When serum creatinine rises to 707 micromol/L, it means that more than 90% of the kidney function is impaired. Renal failure has developed to the end stage, that is, uremia. Therefore, the elevation of serum creatinine for patients with nephropathy is not only the elevation of indicators, but also the fear and fear of uremia.
The rise of serum creatinine is directly related to the impairment of renal function. In the course of the development of nephropathy, there are also some factors that can accelerate the rise of creatinine, such as various infections, misuse of nephrotoxic drugs, high blood pressure, etc. In addition, inappropriate diet will also affect the stability of creatinine, and some dietary habits are not conducive to the stability of renal function, leading to the rise of creatinine.
To avoid creatinine elevation and accelerate renal failure, these four dietary principles should be adhered to:
Serum creatinine is a metabolic product of human muscles. Generally, people with developed muscles have slightly higher serum creatinine than those with less muscles. In general, creatinine level is not affected by diet, but for kidney friends with renal insufficiency, can not metabolize excess creatinine in time, unreasonable diet will affect the creatinine value.
Meat food contains more protein, fat and cholesterol. Too much intake will aggravate the burden of kidney, affect the metabolism of kidney function, and is not conducive to the excretion of creatinine. In addition, creatinine will also be produced in the process of human consumption and absorption of food, thus increasing creatinine. Eating less meat, especially high-fat and cholesterol meat such as animal liver, and eating more vegetables and fruits, which contain more cellulose and vitamins, is conducive to accelerating kidney metabolism, but will not increase the burden of the kidney.
Glomerular filtration rate decreased, all aspects of renal function decreased, and metabolic function was worse. A large number of toxins can not be discharged, resulting in increased levels of toxins in patients, in addition to creatinine, but also lead to increased uric acid. By examining the blood routine, we can see that both serum creatinine and uric acid are elevated.
Therefore, nephropathy patients should also pay attention to low purine diet to avoid aggravating the increase of uric acid and increasing the burden of kidney metabolism.
Patients with renal insufficiency are often accompanied by hypertension. The worse the renal function, the higher the blood pressure is. It is mainly due to the disturbance of the function of regulating water and electrolyte in the kidney, which leads to the retention of water and sodium and the increase of blood pressure. In addition, fibrosis and sclerosis occur after the impaired renal function, and the different blood flow in the kidney will aggravate blood pressure. Hypertension, in turn, can aggravate renal ischemia and hypoxia, forming a vicious circle.
Because of electrolyte disturbance in the later stage of nephropathy, hyperkalemia will occur. Too high blood potassium will easily lead to arrhythmia and other cardiac problems. Then other serious complications will accelerate renal failure, which is not conducive to the stability of renal function and creatinine recovery.
Therefore, patients with CKD 3 or 4 should pay attention to the intake of high potassium food. Relatively speaking, crude grains such as buckwheat, corn, sweet potatoes and other potassium-containing elements are higher in staple foods, bananas, oranges, lemons, apricots, plums and nectarines are slightly higher in fruits, spinach, amaranth, coriander, rape, cabbage and other mungleaf vegetables, as well as potatoes, yam potatoes, fresh peas, soybeans and other potassium-containing elements are higher.
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